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Tachycardia is a psychovascular condition, characterized by a normal heart rate. Supraventricular tachycardia (also called SVT) is the term used to refer to the origin of SVT, i.e., superior ventricular values. Supraventricular tachycardia It can be further divided into atria tachycardia (AVNRT), atrioventricular atrial ostium. reentry tachycardia (AVNRT) and atrioventricular reentry tachycardia (AVRT) based on pathophysiology and origin. In most cases, SVT is considered paroxysmal with rare or infrequent episodes and may take from minutes to months. That will be evident in this memorandum. is reentry supraventricular tachycardia At the same time, emphasis is placed on the most important signs and healing options that must be considered for the conclusion of this mental vascular problem.
What are the Secondary Entrances of Supra-Thyroidal Tachycardia?
There are two primary types based on mechanism and impulse guidance of reentry supraventricular tachycardia :
- AV-nodal reentry tachycardia (AVNRT): involves the AV node and the AV nodal job. Both have the opportunity to go suddenly and slowly.
- Atrioventricular reentry tachycardia (AVRT): involves the AV node, atrial myocardium, accessory pathways, and ventricular myocardium.
What are the symptoms of Re-entry Tachycardia Tachycardia?
SVT is generally considered symptomatic. However, some patients do not experience any known episodes (in the case of brief attacks).
- The clinical situation of the patient is very important in all cases and is usually especially present during individual episodes of rare cardiac pit.
- Sudden episodes are usually accompanied by signs of hemodynamic problems, such as light-headedness, chest discomfort, and respiratory Attacks may last from a few seconds to several hours, sometimes up to 12 hours; clinical symptoms in babies with SVT include preparatory or acute oscillations, nutritional problems, lethargy, and occasional lethargy.
- Prolong tachycardia Episodes may also lead to heart failure. Symptoms are more common in people between the ages of 15 and 35; SVT is more common in women than in men. During this stage, the patient frequently checks for signs during the episode, and his psychological examination and study are common.
- Doctor diagnoses reentry supraventricular tachycardia It is based on the patient’s situation as further confirmed by ECGs performed during SVT episodes. The frequency of cardiac contractions typically varies from 160 to 240 passes per minute. During this time, ECG results are as follows tachycardia No recognizable P-golf; P-waves are still not seen in the QRS ensemble and in a 1:1 ratio.
What is the treatment for recurrent episodes of tachycardia?
Decisions regarding treatment must be made after a thorough clinical examination, followed by imaging studies and radiological studies. Some episodes are usually stopped just before treatment begins.
1. the Vagotone Operation
Early introduction of Vagotone operations helps to stop the dish thyroid. These include cold water swallowing, immersion in cold water on the person, unilateral massage of the carotis card, and tactical Valsalva maneuvers.
2. av-knoopblokker
If these tactical exercises are ineffective and the recorded ECG shows a reduced QRS ensemble (indicating corrective adrenal conduction), the use of AV nodes is indicated. These agents block the guidance of the AV node and help prevent interruption of the reentry cycle. In all of these cases, adenosine is generally considered the agent of choice and the dose should be 6 mg w/v (fast bolus) for adults and 0.05-0.1 mg/kg for infants, plus a bolus of 20 mL saline.
3. adenosine
Adenosine can be used if this treatment protocol fails; two more doses of 12 mg Q 5 min can be given. Sometimes, however, it tends to cause short-lived cardiac arrest, i.e., 2-3 seconds. As a candidate, verapamil can be used at doses of 5 mg IV w/v or diltiazem at 0, 25-0, and 35 mg/kg IV.
4. ablation.
When the reentry supraventricular tachycardia Episodes are becoming more frequent and intrusive, and treatment options are limited and consist of prolonged transverse catheterized anti-alhemia and/or radiofrequency ablation. Ablation is usually preferred. However, if this is not acceptable, prophylaxis is equipped with a product called digoxin, which is switched to a non-dihydropyridine calcium channel blocker, beta-blocker, or a combination of both, depending on the patient’s condition. Additionally, you can create a start with a Class IA, Class IC, or Class III anti-Alismica.
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