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A fibrous cortical defect It is a common benign (unhealthy) tumor of bone, affecting up to 40% of children and more common in young adults. It is still known that it is still known, it can be abbreviated fibroma (nof), a term often used when the tumor or lesion is large and penetrates the central lobe of the bone, the meningeal channel. Other definitions used are fibrotoxic, benign fibrous histiocytoma and non-etiogenic. fibroma .
What are the signs of fibrotic cortical defects?
A fibrous cortical defect is a benign bone loss often seen during radiographic or radiographic examinations. It is the development defect where a portion of the bone is filled in fibrous instead of bone tissue. As a result, it is sometimes referred to as non-stagnation. fibroma the inability of a portion of bone to protrude or develop into bone substance. It usually affects members of the metathesis or long bones, such as the femur (thigh) or tibia (lower leg). In most cases, the injury eventually increases, remodeling it and resulting in an impenetrable piece of bone, but sometimes the injury may grow. Babies and young adults often suffer.
A small fibrous cortical defect The condition is usually asymptomatic and can only be noticed incidentally during an x-ray examination. However, if it is large enough to affect about half of a bone amputation, it can cause pain and increase the risk of an unusual fracture.
X-Ray Presentation
Non-ossifying fibromas There is a well-defined limitation to X-rays and they can appear as a single translucent lesion (no single chamber) of elongated shape with a cross section of less than 2 cm, but they can still be multiple (openings in almost all). When the loss is accompanied by other characteristics such as café-au-lait spots, moles, nevi, hypogonadism (having adequate or little sexual hormones), intellectual resignation and other abnormalities of the eyes and mind, Jaffe-Campanacci syndrome is the diagnostic diagnosis called.
When should I be concerned?
Usually, children with few or no signs need not be cured. However, if the fibrous cortical defect a large part of the bone is affected and becomes quite ill, the risk of pathological fractures increases, and prophylactic surgery can be recommended to stabilize the bone.
Here are some images fibrous cortical defect :
This is a normal X-Ray a fibrous cortical defect The top of the leg bone (proximal tibia).
This is a universal transverse X-Ray fibrous cortical defect Upper part of the leg (proximal tibia).
This X-Ray shows the giant lobule fibrous cortical defect Bottom of leg (distal tibia).
The four images show all the different margins of ossification (sclerosis) of the non-osseous fibromas (Ritschl classification). Stage A (upper left) shows a single round lesion with smooth contours. Stage B (upper right) shows the multi-environmental edge of the lesion. Stage C (right of center) shows a partially calcified (forced) lesion. Stadium D (lower right) shows a complete bombeed (sclerotic) lesion.
The image on the left shows a small fi bone. cortical defect in the thigh with a 7-year-old girl. The image on the right shows a more robust non-starting lesion fibroma that protrudes into the cavity of the central bone (medulla) identified in the lower leg of a 12-year-old patient.
It is not overwhelming. fibroma Seen on CT scan. Left (a), small, untrained axial image fibroma (stadium B), prominently present in the upper arm (humerus), with the same lesion in the sagittal and coronal views (B and C, respectively).
These MRI and scintigraphic images are shown. a fibrous cortical defect IV Heterogeneous low signal intensity (a) (b), which increases after insertion of contrast fluid. Images C and D show untrained fibroma found in a 21-year-old male, thigh (proximal femur). Figure E shows a slight accumulation of radioisotopes in the lesion when scanning the bone (images from Warsaw Medical Research Institute, School of Nuclear Medicine, Warsaw, MD, MD).
Treatment of Fibrous Cortical Defects
Most of these lesions are not considered benign and self-illuminated and therefore do not usually require healing, but as noted above, healing is considered necessary and medical consultation should be followed.
Medical Care
Pathological fractures in children are usually treated with plaster casts to prevent intraoperative metathesis trauma. When. the fibrous cortical defect Regardless of the fact that a fracture union may be needed, healing, not saving and bone grafting.
Surgery
Unstable fractures must be treated surgically, but still lose the greatest risk of pathologic fracture. This includes bone curettage with grafting.
Complications of fibrous cortical defect surgery
A possible aggravation of surgery is trauma to the adjacent actual metacithe, which could be potentially abnormal.
Follow-up.
Patients with normal lesions require only one follow-up radiologic examination 6 to 12 months after diagnosis. A fibrous cortical defect Ossification usually occurs during puberty. However, patients with massive lesions should be followed every 4 to 6 months to assess progression of lesion volume. Patients are advised to avoid high-intensity work, especially contact sports, to prevent fractures.
After fracture, the affected limb should be immobilized until radiological signs of regeneration are seen. If the lesion does not regress and the risk of re-fracture is high, shaving and bone grafting may be necessary with or without internal fixation.
Results and Projections
A fibrous cortical defect Regression usually occurs over time. Monitoring is indicated for patients who require shaving and bone grafting. < Span> Patients with normal lesions require only one follow-up radiologic examination 6 to 12 months after diagnosis.
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